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86 Importantly, this neutrophil reprogramming is not restricted by tumor type or species, 7 suggesting that therapeutic strategies aimed at limiting this differentiation could have broad applicability across different tumor types
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이러한 상승작용적 활성은 플레카이니드가 모다피닐 치료의 지속 기간을 강력히 연장시킨다는 사실로 설명될 수 있었다 (실시예 4)
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