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Beyond behavioral outcomes, mechanistic investigations have linked oxytocin treatment to modulation of neurotransmitter systems, including serotonin and dopamine, and to neuroprotective effects, such as reduced expression of the necroptosis markers RIP3 and MLKL in the hippocampus and amygdala of VPA-exposed rats [41]
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Pharmacological inhibition of MELK restricts ferroptosis and the inflammatory response in colitis and colitis-propelled carcinogenesis
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